Medical Treatments for Prostate CancerThere are many different medical treatments for prostate cancer that involve the clinical care of a healthcare professional. These treatments include expectant therapy, surgery, radiation therapy, hormone therapy, and chemotherapy. Expectant therapy is to carefully observe and monitor the prostate cancer. Because prostate cancer cells often spread very slowly, many older men who have the disease may not need more extensive treatment. However, expectant therapy usually includes routine physician examinations, including digital rectal exams and PSA tests. The different types of surgery for prostate cancer are radical prostatectomy - an open-surgery procedure in which the entire prostate gland and surrounding tissue are removed. Transurethral resection of the prostate (TURP) - surgery to remove part of the prostate gland that surrounds the urethra. Cryosurgery - this procedure involves killing the cancer cells by freezing them with a small metal tool placed in the tumor. Side effects of prostate cancer surgery include incontinence and impotence. Incontinence is the inability to control urine and may result in dribbling of urine, especially immediately after surgery. Normal control usually returns within weeks or months after surgery. Impotence is the inability to achieve an erection. For a month, or so, after surgery, most men are not able to get an erection. Eventually, approximately 40 to 60 percent of men will be able to get an erection sufficient for sexual intercourse, but without ejaculation of semen, since removal of the prostate gland prevents that process.Radiation therapy uses high energy rays to kill or shrink cancer cells, and to decrease their ability to divide. Radiation is often used to treat prostate cancer that is still confined to the prostate gland, or has spread only to nearby tissue. If the disease is advanced, radiation may be used to reduce the size of the tumor and to provide relief from symptoms. Possible side effects of radiation for prostate cancer may include diarrhea, with or without blood in the stool, and colitis, problems associated with urination, a degree of impotence (inability to get an erection), which may occur within two years of radiation therapy. The goal of hormone therapy is to lower the level of male hormones in the body, particularly testosterone. Hormone therapy does not cure the cancer, and is often used to treat persons whose cancer has spread or recurred after treatment. Produced mainly in the testicles, testosterone causes prostate cancer cells to grow. Thus, reduced testosterone levels can make the prostate cancer shrink and become less active. Most studies show that hormone therapy works better if it is started early. Chemotherapy is the use of powerful, anti-cancer medications to kill cancer cells.. Hospitalization may be needed to monitor treatment and chemotherapy's side effects. Common side effects of chemotherapy include: nausea and vomiting, hair loss, anemia, reduced ability of blood to clot, mouth sores, increased likelihood of developing infections, fatigue. Most side effects disappear once treatment is stopped. |
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The Biotech Weblog: Snake Venom Toxin Inhibits Human Prostate Add your response to Snake Venom Toxin Inhibits Human Prostate Cancer Cell Growth:. Name: *, Email: (will never be shown)*, URL: Antiproliferative effect on human prostate cancer cells by a Antiproliferative effect on human prostate cancer cells by a stinging nettle root extract - Urtica dioica from Alternative Medicine Review in Health Cannabinoid receptor agonist induced apoptosis of human prostate Cannabinoid receptor agonist induced apoptosis of human prostate cancer cells LNCaP proceeds through sustained activation of ERK1/2 leading to G1 cell cycle Bcl-2 and Bcl-xL differentially protect human prostate cancer Bcl-2 and Bcl-xL differentially protect human prostate cancer cells from induction of apoptosis by melanoma differentiation associated gene-7, mda-7/IL-24 Oncogene - Abstract of article: Diallyl trisulfide-induced Diallyl trisulfide-induced apoptosis in human prostate cancer cells involves c-Jun Using PC-3 and DU145 human prostate cancer cells as a model, Resveratrol-induced gene expression profiles in human prostate Resveratrol-induced gene expression profiles in human prostate cancer cells. Sunita B JONES, Samuel E DEPRIMO, Michael L WHITFIELD, James D BROOKS Cancer Pepper component hot enough to trigger suicide in prostate cancer "Capsaicin had a profound anti-proliferative effect on human prostate cancer cells in culture," said Sören Lehmann, M.D., Ph.D., visiting scientist at the Green Tea Plus Painkiller Slows Prostate Cancer Cell Growth In their current studies, treatment of cultured human prostate cancer cells with either a COX-2 inhibitor or green tea extract significantly inhibited IngentaConnect beta-Lapachone-Induced Apoptosis in Human Prostate Here we show that NAD(P)H:quinone oxidoreductase (NQO1/xip3) expression in human prostate cancer cells is a key determinant for apoptosis and lethality The Progression of LNCaP Human Prostate Cancer Cells to Androgen The Progression of LNCaP Human Prostate Cancer Cells to Androgen Independence Involves Decreased FOXO3a Expression and Reduced p27KIP1 Promoter The Progression of LNCaP Human Prostate Cancer Cells to Androgen Soy Isoflavones Exert Differential Effects on Androgen Responsive Genes in LNCaP Human Prostate Cancer Cells J. Nutr., April 1, 2007; 137(4): 964 - 972. IngentaConnect Transdifferentiation of Cultured Human Prostate Transdifferentiation of Cultured Human Prostate Cancer Cells to a Neuroendocrine Cell Phenotype in a Hormone-Depleted Medium. Authors: Shen R.; Dorai T.; Suppression of Human Prostate Cancer Cell Growth By {{alpha}}1 1-adrenoceptor antagonists was examined in two human prostate cancer cell lines, PC-3 and DU-145, and a prostate smooth muscle cell primary culture, SMC-1, PCPH/ENTPD5 Expression Enhances the Invasiveness of Human Prostate PCPH/ENTPD5 Expression Enhances the Invasiveness of Human Prostate Cancer Cells by a Protein Kinase C {delta} â“Dependent Mechanism Unique Grape Skin Extract Inhibits Prostate Cancer Cell Growth in Using a series of human prostate cancer cells, representing different stages of prostate cancer progression, the researchers showed that MSKE significantly Pectin induces apoptosis in human prostate cancer cells Pectin induces apoptosis in human prostate cancer cells: correlation of apoptotic function with pectin structure. Crystal L Jackson2,3, Tina M Dreaden2,3, Antiproliferative effect on human prostate cancer cells by a Antiproliferative effect on human prostate cancer cells by a stinging nettle root (Urtica dioica) extract. Konrad L, Muller HH, Lenz C, Laubinger H, The human prostate cancer cell line LNCaP bears functional The human prostate cancer cell line LNCaP bears functional membrane testosterone receptors that increase PSA secretion and modify actin cytoskeleton. |
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